Scientists are digging into the workings of the endogenous cannabinoid system — and finding it helps regulate a multitude of biological functions.
terminals pass chemical signals along to other neurons, while, at the other end, dendrites receive chemical signals from other neurons. The space between an axon terminal and a dendrite, where chemical signals are deposited and picked up, is called a synapse.
At least, that’s what you'll learn from the neuroscience unit of any psychology 101 course. For the most part it’s true. But the ECS is an exception to the rule. While the majority of neurotransmitters are marching along in one direction from neuron to neuron, cannabinoids refuse to step in line. They travel across the synapse inThis difference in design hints at one of the central functions of the ECS: It regulates the strength of neurological signals.
Interestingly, THC is a weak agonist of CB1, meaning it is relatively clumsy at binding to the receptor. This is where many explanations of how cannabis gets users “high” go awry. While many simple explanations put forth that THC simply floods cannabinoid receptors throughout the body kicking them into overdrive, the truth is more nuanced.
“It’s like in a workplace. There’s a certain number of chairs in the room,” Lu says. “You could have all good employees getting the work done effectively. Then when you have some less good employees coming in and occupying positions the overall efficiency is reduced.” On the other hand, if there are few other cannabinoids around, THC will clumsily bind to CB1 receptors causing them to be more active. A few mediocre workers is better than a room full of empty chairs.
Of course, since THC is a second-rate CB1 agonist, another question emerges: What are the effects of aCB1 agonist? The answer isn’t pretty. Beginning in the late 2000s, a drug colloquially known as “spice”
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