Does an ancient virus genome drive autism? MolPsychiatry
Now, an international research collaboration including Kobe University's Professor Takumi Toru and Researcher Chia-wen Lin and colleagues have made new discoveries regarding autism onset in mouse models.
Fig. 2. a–c. Comparison of the repeated sequence of copy number variations, including retrovirus genes on the genome. d and e. Gene expression level analysis of repeated sequences: BTBR mice had more copy numbers of the endogenous retrovirus gene ERV than normal mice, and a portion of these genes were activated in BTBR mice. Credit:Autism is a neurodevelopmental disorder that remains largely unexplored despite the rapidly increasing number of patients.
Fig. 3. a. AGM. B. Yolk sac: For BTBR/R autism model mice, genes with increased expression are indicated in pink and genes with decreased expression are in light blue. c–f. Comparison of gene expression levels in each cluster. In BTBR/R mice, there were changes in the expression of various genes that are indicative of endogenous retrovirus activation.
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