A Japanese billionaire recruits an artists-only crew for a space voyage around the moon.
A commercial DJ, K-pop rapper, and a space YouTuber are to go on a trip around the Moon, after they were picked by a Japanese billionaire for a private SpaceX flight.
Businessman Yusaku Maezawa revealed his crew on Friday after a global search for creative individuals last year.The flight, scheduled for next year, could be the first lunar orbital journey by humans since 1972. The proposed fly-by would see a spacecraft circle the moon, coming within 200km of the surface. The trip would take eight days from launch to return.the Starship rocket from Elon Musk's SpaceX companyThe ship has not even been approved for an orbital journey around the Earth and has been grounded for the past 18 months in Texas, afterBut Mr Maezawa made no mention of this delay in his video announcing the crew for the mission he's dubbed dearMoon.
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Novel STAT1 Variants in Japanese Patients with Isolated Mendelian Susceptibility to Mycobacterial Diseases - Journal of Clinical ImmunologyPurpose Heterozygous dominant-negative (DN) STAT1 variants are responsible for autosomal dominant (AD) Mendelian susceptibility to mycobacterial disease (MSMD). In this paper, we describe eight MSMD cases from four kindreds in Japan. Methods An inborn error of immunity-related gene panel sequencing was performed using genomic DNA extracted from whole blood samples. The identified variants were validated using Sanger sequencing. Functional analysis was evaluated with a luciferase reporter assay and co-transfection assay in STAT1-deficient cells. Results Patient 1.1 was a 20-month-old boy with multifocal osteomyelitis and paravertebral abscesses caused by Mycobacterium bovis bacillus Calmette-Guérin (BCG). Although the paravertebral abscess was refractory to antimycobacterial drugs, the addition of IFN-γ and drainage of the abscess were effective. Intriguingly, his mother (patient 1.2) showed an uneventful clinical course except for treatment-responsive tuberculous spondylitis during adulthood. Patient 2.1 was an 8-month-old boy with lymphadenopathy and lung nodules caused by BCG. He responded well to antimycobacterial drugs. His mother (patient 2.2) was healthy. Patient 3.1 was a 11-year-old girl with suspected skin tuberculosis. Her brother (patient 3.2) had BCG-osis, but their mother (patient 3.3) was healthy. Patient 4 was an 8-month-old girl with left axillary and supraclavicular lymphadenopathy associated with BCG vaccination. Kindreds 1, 2, and 3 were shown to have novel heterozygous variants (V642F, R588C, and R649G) in STAT1, respectively. Kindred 4 had previously reported heterozygous variants (Q463H). A luciferase reporter assay in STAT1-deficient cells followed by IFN-γ stimulation confirmed that these variants are loss-of-function. In addition, with co-transfection assay, we confirmed all of these variants had DN effect on WT STAT1. Conclusion Four kindred MSMD subjects with 3 novel variants and 1 known variant in STAT1 were identified in this study. AD S
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